A bal oldali lateralis májlebeny csavarodása shagya arab kancában - Esetismertetés

Abstract

SUMMARY Background: Liver lobe torsion is a very remarkable disorder in equine patients. It is diagnosed infrequently, as only eight cases have been reported so far in the literature. Its clinical signs are non-specific, definitive diagnosis is usually reached through exploratory laparotomy. The resection of the affected lobe via stapler device was successful in five cases. Materials and Methods: the aim of the current article is to report clinical his-tory and necropsy findings in a horse with torsion of the left lateral liver lobe, and also to review the characteristics of the mentioned condition.Results and Discussion:this case report presents a 9 years old, Shagya Ara-bian mare (6 months pregnant) with a history of inappetence, lethargy, mild colic signs, and tachycardia. At admission the horse showed signs of lethargy. Physi-cal evaluation revealed tachycardia (80 beats/min), tachypnoe (30 breaths/min.)and dirty red mucous membranes with petechial haemorrhages (CRT: 3 sec.). There was total absence of gut sounds. No reflux was obtained. Findings on rectal examination were within normal limits. The peritoneal fluid collected via abdominocentesis was grossly serosanguineous (WBC count: 10.53 G/l; total pro-tein: 34 g/l; lactate: 6.8 mmol/l). Blood haematological and biochemical analysis was performed. The PCV was moderately elevated (55.56%), thrombocytopenia (35 G/l), mild leucocytosis (13.07 G/l) with neutrophilia, decreased total protein (50 g/l) and critically elevated lactate concentration (13.1 mmol/l) was evident. Exploratory laparotomy was performed. The pre-operative treatment included intravenous fluid therapy and parenterally administered flunixin-meglumine (1.1mg/kg) and butorphanol (20 mg). The mare could not tolerate general anaesthe-sia and died soon after the surgical incision. Serosanguineous peritoneal fluid, petechial haemorrhages on the visceral and on the parietal peritoneum indicat-ing endotoxin shock was found post mortem. The necropsy revealed that the left lateral lobe of the liver was twisted. The twisted lobe was diffusely necrotic and congestion was also evident. No other possible causes of endotoxin shock could be revealed.