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A tendon prepubicus szakadása vemhes kancában: két klinikai eset bemutatása

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643_650_Farkas_ló.pdf (1.449Mb)
Date
2024-11
Author
Farkas, Ágnes Klára
Horti, Klára
Tóth, Balázs
DOI link
10.56385/magyallorv.2024.11.643-650
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Abstract
Background: The prepubic tendon rupture is a rare condition of the late pregnant mare. The condition is difficult to manage, and results in high-risk pregnancy with dystocia. Rupture of the prepubic tendon in mare is not a surgical condition. Mares with complete rupture of the prepubic tendon carry a poor prognosis. Preterm foals have grave prognosis for survival, while near term foals may survive with intensive care. Objectives: The aim of the current report was to review the published data and describe the clincal findings, treatment and outcome of horses diagnosed with pre- pubic tendon rupture. Materials and Methods: Two horses were diagnosed with prepubic tendon rupture based on history, physical examination and abdominal ultrasonography. The horses were treated conservatively with stall rest, abdominal support, antiinflammatories and opioids. One case was referred at the Department and Clinic of Equine Medi- cine, University of Veterinary Medicine Budapest. The other mare was treated on-site. Results and Discussion: The mares started to show clinical signs at the end of the last trimester (316 and 335 days). The mares were reluctant to move and showed mild colic sings. The heart rate was elevated in both cases (68 bpm and 72 bpm). The mares exhibited substantial ventral edema extending from the udder to the xyphoid cartilage. During the abdominal ultrasound examination, sonographic signs of marked interstitial fluid accumulation were visible in the caudal part of the rectus abdominis muscle in both cases. In one case, bloody exudate was oozing from the teats. The horses went into labour spontaneously at day 318 and 340 of pregnancy with apparent dystocia and assistance was required during foaling. Both foals were still-born. The mares were euthanased few days after the parturition because of uncontrollable pain, progression of rupture and circulatory decompensation.
URI
http://hdl.handle.net/10832/4123
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