| dc.contributor.author | Michelberger, Vincent | |
| dc.date.accessioned | 2025-05-08T06:26:59Z | |
| dc.date.available | 2025-05-08T06:26:59Z | |
| dc.date.issued | 2024 | |
| dc.identifier.uri | http://hdl.handle.net/10832/4379 | |
| dc.description.abstract | The p53 protein family is pivotal in preventing tumoral cell transformations by aiding DNA
repair or inducing apoptosis in cases of irreparable damage. This mechanism activates not
only for tumor prevention but also for various DNA damages, such as those caused by
oxidative stress from inhalation of Zn or Cu oxides. These oxides can induce metal fume
fever (MFF), an occupational ailment primarily affecting welders and metal workers,
potentially leading to chronic asthma. | en_US |
| dc.language.iso | en | en_US |
| dc.title | Measurement of the gene expression rate of certain p53 family genes in experimental animals after metal oxide fume inhalation. | en_US |
| dc.title.alternative | The p53 gene family activation after metal oxide fume inhalation in experimental animals and the investigation of the role of these genes in the pathophysiology of the disease called metal fume fever. | en_US |
| dc.type | Thesis | en_US |
