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dc.contributor.authorJakab, Szilvia
dc.contributor.authorBali, Krisztina
dc.contributor.authorHomonnay, Zalán
dc.contributor.authorKaszab, Eszter
dc.contributor.authorIhász, Katalin
dc.contributor.authorFehér, Enikő
dc.contributor.authorMató, Tamás
dc.contributor.authorKiss, István
dc.contributor.authorPalya, Vilmos
dc.contributor.authorBányai, Krisztián
dc.date.accessioned2024-09-23T08:19:16Z
dc.date.available2024-09-23T08:19:16Z
dc.date.issued2023
dc.identifier.citationJakab S, Bali K, Homonnay Z, Kaszab E, Ihász K, Fehér E, Mató T, Kiss I, Palya V, Bányai K. Genomic Epidemiology and Evolution of Fowl Adenovirus 1. Animals (Basel). 2023 Sep 5;13(18):2819. doi: 10.3390/ani13182819en_US
dc.identifier.urihttp://hdl.handle.net/10832/4074
dc.description.abstractFowl adenovirus 1 (FAdV-1) is the main cause of gizzard erosion in chickens. Whole genome sequencing and sequence analyses of 32 FAdV-1 strains from a global collection provided evidence that multiple recombination events have occurred along the entire genome. In gene-wise phylogenies, only the adenoviral pol gene formed a tree topology that corresponded to whole genome-based phylogeny. Virus genetic features that were clearly connected to gizzard erosion were not identified in our analyses. However, some genome variants tended to be more frequently identified from birds with gizzard erosion and strains isolated from healthy birds or birds with non-specific pathologies tended to form common clusters in multiple gene phylogenies. Our data show that the genetic diversity is greater, and the evolutionary mechanisms are more complex within FAdV-1 than previously thought. The implications of these findings for viral pathogenesis and epidemiology await further investigation.en_US
dc.language.isoenen_US
dc.titleGenomic Epidemiology and Evolution of Fowl Adenovirus 1en_US
dc.typeArticleen_US
dc.identifier.doi10.3390/ani13182819


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